Info

🌱 來自: Etiologies-hyperaldosteronism

Nonaldosterone mineralocorticoid excess

• 11β-HSD defic. (→ lack of inactivation of cortisol, which binds to mineralocorticoid recept.)
• Liddle’s syndrome (constitutively activated/overexpressed distal tubular renal Na channel)
• Black licorice (glycyrrhizic acid inhibits 11β-HSD), extreme hypercortisolism (overwhelming 11β-HSD), exogenous mineralocorticoids