ST-elevation
(NEJM 2003;349:2128; Circ 2009;119:e241 & e262)
參考 ➡️ ST-segment elevation in conditions other than acute myocardial infarction
-
Acute MI: upward convexity STE (ie, a “frown”) ± TWI (or prior MI w/ persistent STE)
-
Coronary spasm: Prinzmetal’s angina; transient STE in a coronary distribution
-
Pericarditis: diffuse, upward concavity STE (ie, a “smile”); a/w PR ↓; Tw usually upright
-
HCM, Takotsubo CMP, ventricular aneurysm, cardiac contusion
-
Pulmonary embolism: occ. STE V1–V3; classically a/w TWI V1–V4, RAD, RBBB, S1Q3T3
-
Repolarization abnormalities:
LBBB (↑ QRS duration, STE discordant from QRS complex; see “ACS” for dx MI in LBBB)
LVH (↑ QRS amplitude); Brugada syndrome (rSR′, downsloping STE V1–V2); pacing
Hyperkalemia (↑ QRS duration, tall Ts, no P’s); epsilon waves (late afterdepol.) in ARVC
-
aVR: STE >1 mm a/w ↑ mortality in STEMI; STE aVR > V1 a/w left main disease
-
Early repolarization: most often seen in V2–V5 in young adults (Circ 2016;133:1520)
1–4 mm elev of notch peak or start of slurred downstroke of R wave (ie, J point); ± up concavity of ST & large Tw (∴ ratio of STE/T wave <25%; may disappear w/ exercise)
? early repol in inf leads may be a/w ↑ risk of VF (NEJM 2009;361:2529; Circ 2011;124:2208)
- Post-ROSC: transient STE can be seen w/in 1st ~8 mins; not indicative of ACS