Info

🌱 來自: Huppert’s Notes

Quantitative Platelet Disorders🚧 施工中

Quantitative Platelet Disorders

Thrombocytopenia

•   Definition*:* Low number of platelets (<150K/μl)

•   Symptoms*:* Easy bruising or bleeding (e.g., mucosal bleeding such as gingiva or nasal mucosa, heavy menses, petechiae)

•   Work-up*:*

-   Get a peripheral blood smear:

   Rule out pseudothrombocytopenia (platelet clumping), which can occur in the blood tube when the anticoagulant EDTA is present, causing a spuriously low platelet count. Evaluate by looking for platelet clumping on the blood smear. Resend platelets in a heparin or sodium citrate tube if needed.

   Giant platelets can be falsely counted by the automated cell counter as WBCs or RBCs. Large platelets can occur with inherited platelet disorders.

-   Take a thorough history:

   History of easy bleeding or bruising?

   Systemic symptoms – fever, headache, abdominal pain, hematuria, diarrhea? These symptoms can occur with HUS or TTP.

   Medications? In the hospital, consider heparin products, antibiotics, immunosuppressants.

   History of autoimmune conditions? A history of autoimmune conditions makes ITP more likely.

-   Also check PT, PTT, reticulocyte count, fibrinogen, LDH, TSH, HIV, HCV, and other labs guided by the disease entity suspected

•   Etiologies of thrombocytopenia: Consider three buckets:

-   Decreased production: Think about processes that cause bone marrow suppression or infiltration. Often multiple cell lines down!

   Aplastic anemia, MDS, leukemia, Fanconi syndrome, infiltrative disorders

   Medications/toxins: Alcohol, chemotherapeutic agents, chloramphenicol, benzene, radiation

   Infections: Parvovirus, EBV, CMV, HCV, HIV

   Nutritional deficiencies: B12 or folate deficiency

-   Increased destruction: Divide causes by immune-mediated or non-immune mediated. Also see additional details about these conditions in the section below.

   Immune-mediated

-   Primary: ITP

-   Secondary: HIT, other medications (antibiotics, H2 blockers), infections, lupus, APS, lymphoproliferative disease

   Non-immune-mediated

-   Microangiopathic hemolytic anemia (MAHA): HUS, TTP, DIC, scleroderma renal crisis

-   HELLP

-   Mechanical: CVVH

-   Sequestration/dilutional

   Hepatosplenomegaly: Cirrhosis and portal HTN can sequester 90% of platelets

   Massive transfusion: 10U pRBCs decrease platelets by 50%